Category Archives: Birth Trauma

Respecting the 4th trimester and your postpartum body …

Respecting your body in the 4th trimester …. we read so much about the 1st, 2nd & 3rd trimesters of what to expect … however have you considered the 4th trimester and what YOU need to have in place to support you?
Your body is expected to do this amazing feat … to feed your baby, to do nights which might be very disturbed … to get out there and get back in shape (hey what???) yup the pressure women place on themselves that they need to lose the baby weight and get back into those pre-pregnancy jeans asap is immense.

In my role as a doula and pregnancy & postpartum yoga teacher … I have heard some scary stuff … I need to get back to where I was pre-pregnancy … I need to start running …. STOP 

Consider this … your body has just worked exceptionally hard for 9 months nurturing & nourishing a baby … your pelvic floor has carried your growing baby(ies) … you have these amazing hormones whizzing around your body … why do you have to jump back onto the hamster wheel?

I see ladies come along to class struggling with discomfort in their pelvis, pain in their lower backs, misaligned pelvises, there pelvic floor feels like it is non-existent, they haven’t eaten breakfast or been able to have a hot drink … they are struggling from having had a c-section … remember this is major abdominal surgery that can take quite a while to heal from … you want to feel that what you are engaging in is nurturing and nourishing you … yes there will come a point when you feel that you can start that “buggy fit” class and that you are ready …. but what happens x number of months, years down the road when you are suffering with pelvic leakage, prolapse …. this is the time to gently reconnect with your body, to be gentle and learn what it can do and what it needs … nurture and nourish your pelvic floor ready for your next baby or for the next stage of your journey.  Just because x or y says you should do it doesn’t mean it’s right for you at this moment … take the space to nurture yourself until your baby is at least 6 months … respect your body, respect the amazing concoction of hormones.  

There are many cultures who honour the postpartum woman … with periods of lying in … special foods, herbs, treatments to keep the body warm and nourished, massage … sounds divine … I so wish when I had had my babies that I had had this … after trying to get up and out after having an amazing VBAC birth with our second baby I ended up in the local supermarket bumping into someone who had only that morning opened the email to say “M had arrived earth side … welcome & all were well and doing fine” … imagine the shock on her face to see me coming through the doors … she was catching flies … I must add that I struggled to do what I needed to do and ended up going home and doing an online shop and promising myself that I was going to be gentle … it was too soon and too much … I ended up online shopping and taking a couple of weeks to myself … I learnt the hard way … and I chat about this a lot in class … but again social expectations are that we should be bouncing back and up at it … and also the pressure we put ourselves under.  

Take the six weeks to connect with your baby, start those gentle breaths we connect with in class … reconnect with your abdominal muscles and pelvic floor, respect your body, your baby but ultimately respect YOURSELF … you are enough and you do not need to be super woman … your are nourishing & nurturing yourself and a baby … enjoy … have those pyjama days, snuggle with your baby, don’t pack your days running form an activity in the morning, lunch with a, b & c and then another activity in the afternoon.  Pace yourself … have space to spend time at home … to enjoy your baby … connect to what your body needs … 


Claire runs Well Woman Happy Baby classes … she is based in Northampton from The Space in Boothville.  She has over 15 years of supporting women and is passionate about pregnancy and the postpartum period.  The classes are a combination of postnatal mummy yoga to realign and reconnect, baby massage & baby yoga plus a whole host of different techniques to support you and your baby.  There is a always a cuppa and some treat  that is created by Claire … plus the amazing support of those in class with you.  Classes are kept to a maximum of 4 mummies and babies.  Very bespoke to you and your little one.  
Claire encouragés you to listen to your baby, to journey together and if you need to baby wear in class she will adapt the class to facilitate this and it might just surprise you that you will do a relaxation with your baby fast asleep on your chest and it will be the most amazing experience … come and explore but be prepared to be here for a good few months … so build this into your postpartum care budget … it will be money well spent on you both.  Happy mummy, happy baby = happy daddy.

The ladies and their babies who come along really benefit from these amazing classes … 

“I found these classes invaluable my pelvic floor took a real hammering in labour and I have journey for 7 months working really hard to gain stability, balance and strength back.  I couldn’t have done that without your classes …. thank you I am very grateful!”

“I find the WWHB invaluable! And H loves them!”

“Please don’t stop ….. the progression from pregnancy yoga to WWHB was fab for us, J loved it and invaluable for me taking those first steps into mummy-life xx”

“Definitely keep them, I loved them with Z and N!”

“These sessions have been so important to me! I love them and have had a welcoming and safe place to come every week. I tell everyone I meet!”

“I can’t wait to have another baby just so I can come back to you Claire!”


Evidence for the Vitamin K shot


This is so informative and yes I have brought the information across with full references etc and citations to the author including links to her interviews about writing this essential information on Vitamin K.  

If you wish me to remove it … I will but please please keep the page live for as long as possible as this is such important information that parents really struggle to find … thank you Cx 

Evidence for the Vitamin K Shot in Newborns

March 18, 2014 by Rebecca Dekker, PhD, RN, APRN
© Copyright Evidence Based Birth. Please see disclaimer and terms of use.

Vitamin K deficiency bleeding, thought to be a problem of the past—has been recently thrust back into the spotlight. During an 8-month period in 2013, five infants were admitted to Vanderbilt Children’s Hospital in Nashville, Tennessee, with life-threatening bleeding. The infants were diagnosed with late Vitamin K deficiency bleeding (VKDB)—four of the infants had bleeding in the brain, and one had bleeding in the intestines. Although the five infants survived, two required emergency brain surgery to save their lives, one has severe brain damage (a stroke with right-sided paralysis and severe cognitive delays), and two have mild to moderate brain injuries (Personal communication, Dr. Robert Sidonio, 2014).

What did these infants have in common? The infants ranged in age from seven weeks to five months old; three were male and two were female. Three of the infants were born in hospitals, and two were born at home. All of them had uncomplicated, spontaneous vaginal births (no forceps, no vacuum, no C-sections). All of the infants were exclusively breastfed. Most importantly, what these infants had in common was that all of their parents haddeclined Vitamin K shots at birth.

Concerned by this outbreak, the hospital asked the Centers for Disease Control (CDC) to look into the situation. Researchers from the CDC examined Tennessee hospital records and found that between the years 2007 and 2012, there had been zero cases of Vitamin K deficiency bleeding out of more than 490,000 births. They randomly sampled records from babies born at three Nashville hospitals and found that 96.6% of infants received Vitamin K injections. In contrast, only 72% of infants born in local freestanding birth centers received Vitamin K (Warren, Miller et al. 2013).

When the parents of the five infants were asked why they had declined Vitamin K, their reasons for declining included: concern about an increased risk for leukemia, a belief that the injection was unnecessary and “unnatural,” and a fear that their infant would be exposed to toxins in the shot. Only one of the families was aware that life-threatening bleeding was a possibility if they declined the injection (Warren, Miller et al. 2013; Personal communication, Dr. Robert Sidonio, 2014).

So what is the deal with Vitamin K? Why do most babies receive a Vitamin K shot? Can the shot really cause leukemia? (The answer is no.) What are the chances that an infant will develop life-threatening bleeding if he does not receive the Vitamin K shot?  What is the evidence that the shot prevents bleeding? Are there any alternatives? These are the questions we will be tackling in this Evidence Based Birth article.

Note: This article includes photos and a story of an infant who experienced late Vitamin K deficiency bleeding. The purpose of this story is not to sway you to make a decision one way or another. If you have ever read any of my other articles, or taken one of my Evidence Based Birth classes, you will know that one of my teaching methods is to use real-life stories to teach about health conditions. Although Vitamin K deficiency bleeding is rare, it is real and it can happen to real infants. As always, please read the Evidence Based Birth disclaimer (below) and do not make any health care decisions without talking with your care provider first.

Evidence Based Birth Disclaimer

What is Vitamin K and what does it do in the body?

Vitamin K is a fat-soluble vitamin needed for blood clotting. It is named after the German word for clotting—Koagulation. We cannot make Vitamin K ourselves, and we don’t store it very well in our body. We get Vitamin K1 (also known as phyllpJ?pJ?? ?@???J??J??J? that live in our intestinal tracts.

Vitamin K1 from plants makes up about 90% of our overall Vitamin K levels, while Vitamin K2 from bacteria makes up only about 10% of our overall Vitamin K intake. (Shearer 2009Lippi and Franchini 2011; Polin, Fox et al. 2011)


Olive was born in a birth center. Her Vitamin K shot was unintentionally omitted at birth, and she suffered a severe brain bleed at 2 months old. Photos used here with permission from Olive's parents.


Foods that are rich in Vitamin K1 include:

  • Leafy green vegetables, such as spinach, kale, and swiss chard
  • Cabbage
  • Cauliflower
  • Broccoli
  • Turnips
  • Brussels sprouts
  • Avocado
  • Banana
  • Kiwi
  • Soybean oil

Vitamin K is necessary for our bodies to activate certain molecules (also known as clotting factors) that help the blood to clot. The blood clotting factors are there in normal numbers at birth, but not activated fully due to low levels of Vitamin K.  If we do not have enough Vitamin K, then we cannot activate these molecules. So a Vitamin K deficiency makes our blood less able to clot.

For the most part, our bodies can continue to clot appropriately with low Vitamin K levels. However, as the levels get lower and lower, we can suddenly reach a point where our bodies can no longer clot and we start to bleed spontaneously.  The level that you have to get down to for bleeding to start varies from person to person.  When the bleeding will occur is completely unpredictable (Shearer 2009).

What can happen if a baby does not have enough Vitamin K?

A baby who does not have enough Vitamin K can start to bleed suddenly, without warning. This is known as Vitamin K deficiency bleeding.

Vitamin K deficiency bleeding can be idiopathic or secondary.

  • Idiopathic VKDB means that the cause is unknown. Virtually all cases of idiopathic VKDB happen in babies who are exclusively breastfed (Shearer 2009).
  • Secondary VKDB means that the baby has an underlying disorder such as gallbladder disease, cystic fibrosis, or medication side effects. Most babies who have secondary VKDB are also exclusively breastfed (Shearer 2009)..

Vitamin K deficiency bleeding can follow one of three patterns: early, classical, and late.

  • Early VKDB happens in the first 24 hours of life. Early VKDB is usually seen in babies born to mothers who took medicines that interfere with Vitamin K. These medicines may include warfarin (Coumadin), seizure medications, and tuberculosis medications. The bleeding usually happens in the skin, brain, and abdomen (Shearer 2009).
  • Classical VKDB happens in days 2-7 of life, usually during days 2-3. This is when levels of Vitamin K are lowest. Common bleeding sites include the gastrointestinal system, umbilical cord site, skin, nose, and circumcision site. The official cause of classical VKDB is listed as “unknown,” but breastfeeding and poor feeding (<100 mL milk/day or <3.4 ounces milk/day) are major risk factors (Shearer 2009).
  • Late VKDB happens after the first week of life, usually during weeks 3-8. The bleeding usually happens in the brain, skin, and gastrointestinal tract. Bleeding in the brain is often the first sign of late VKDB. Late VKDB happens in exclusively breastfed infants who did not receive a Vitamin K shot. Some infants may also be at higher risk if they have undetected gallbladder disease, cystic fibrosis, chronic diarrhea, and antibiotic use. (Shearer 2009)

What is the history of giving Vitamin K to newborns?

  • In 1894, a Boston physician named Dr. Townsend described 50 cases of bleeding in newborns. He called these cases “Haemorrhagic Disease of the Newborn (HDN).” Dr. Townsend was the first person to figure out that there was a connection between poor or insufficient breastfeeding and bleeding in newborns (Shearer 2009).
  • In 1930, a Danish biochemist found that Vitamin K deficiency was the cause of unexpected bleeding in baby chicks (Lippi and Franchini 2011).
  • In 1944, a definitive Swedish study was published including more than 13,000 infants who were given 0.5 mg of Vitamin K (either oral or injection) on the first day of life. The researcher found that infants who received Vitamin K experienced a 5-fold reduction in the risk of bleeding to death during the first week of life. It was estimated that for every 100,000 full-term infants who were born, Vitamin K would save the lives of 160 infants per year (Lehmann 1944). See Table 1, below.
  • In 1961, after nearly 2 decades of research had been published, the American Academy of Pediatrics recommended giving Vitamin K shots after birth. This practice has been the standard of care in the U.S. ever since (AAP, 1961).
  • By 1999, the name HDN was changed to Vitamin K deficiency bleeding (VKDB) to indicate that that this condition is caused solely by Vitamin K deficiency. The new name also reflects the fact that some infants begin bleeding later in infancy—after the four-week newborn period is over (Shearer 2009).

Table 1: Newborn deaths due to bleeding before and after Vitamin K was introduced on September 1st, 1940 (Lehmann, 1944).


Total infants

Infant deaths from bleeding on:

1st day 2nd day 3rd day 4-8th day Total 2-8thday
September 1st 1934 to August 31st, 1937






16 (2%)

September 1st, 1937 to August 31st, 1940






18 (1.84%)

September 1st, 1940 to August 31st, 1943

13, 250





6 (0.45%)


How common is late Vitamin K deficiency bleeding (Late VKDB)?

Late bleeding (after the first week of life) is the most dangerous kind of VKDB (Shearer 2009).

  • When infants do not receive any Vitamin K at birth, statistics from Europe show that 4.4 to 10.5 infants out of 100,000 will develop late VKDB. Rates are higher in Asian countries (1 out of every 6,000 infants).
  • When infants receive oral Vitamin K at least three times during infancy (typically at birth, one week, and four weeks), anywhere from 1.4 to 6.4 infants out of 100,000 will develop late VKDB.
  • When infants receive the Vitamin K shot at birth, there are virtually no cases of late VKDB.

For references for these numbers, see the Table at the bottom of the article.

Overall, late VKDB is rare in most European countries. On the other hand, it is more common in some Asian countries, such as Japan, Vietnam, and Thailand. In Thailand, back in the 1980s, infants did not receive any Vitamin K at birth. At that time, researchers reported that 72 out of 100,000 infants developed late VKDB. Researchers don’t know why rates of late VKDB are higher in some countries compared to others, but it’s thought that it is related to a combination of environment (diet) and genetics (Hanawa, Maki et al. 1988Danielsson, Hoa et al. 2004).

The U.S. does not track the number of cases of late VKDB, so we don’t really know how frequently it would occur if we stopped giving the shot routinely.

What are the potential consequences of late VKDB?

Although late VKDB is rare, the consequences can be catastrophic. More than half of infants who develop late VKDB will have bleeding in the brain. The mortality rate for late VKDB is approximately 20%. (Shearer 2009Lippi and Franchini, 2011). One study that looked at 131 cases around the world found an overall death rate of 14%. Of the surviving infants, about 40% had long-term brain damage.

In low-income countries, many babies with late VKDB may die before reaching the hospital, and because their diagnoses and deaths are undocumented, these cases would not be counted in any of the VKDB statistics. (Shearer 2009)

One of the most dangerous things about VKDB is that you may not recognize it until it is too late. Infants cannot tell us what is going on, and symptoms of brain injury may be subtle— such as difficulty feeding, lethargy, or fussiness. Unfortunately, a brain bleed may reach a critical size before parents seek medical attention. And it can take even longer for health care professionals to figure out what is wrong.

“Olive had struggled to eat the night before, so she and I had been up all night. When she seemed absolutely exhausted the next day, I chalked it up to the previous night’s sleeplessness and didn’t think much of it. I decided we would take a nap and see if that solved anything, and call the doctor if it didn’t. When we woke up, Olive could barely open her eyes – the only word I could think of was “lethargic,” and since that never coincides with anything good, I immediately called the doctor. I explained what had been happening and they had me take her temperature, which turned out to be 96.7 degrees. She hadn’t felt feverish, so I didn’t take a temperature before then – turns out a low temperature is just as bad. The doctor told us to go to the ER immediately…”

~Stefani, mother of Olive, who had late VKDB when she was 2 months old

Read more of Olive’s story here: Part 1Part 2, and Why it Happened

What about classical VKDB? Is that dangerous?

Classical VKDB—bleeding that occurs in the first week of life—is more common than late VKDB. Information from older studies shows that 0.25% to 1.5% of newborns (250 to 1,500 out of 100,000) will experience classical VKDB if they don’t receive Vitamin K at birth, while more recent reviews show that the number is closer to 0 to 0.44% (0 to 440 out of 100,000). This type of VKDB is usually mild and involves bleeding at the umbilical cord site or circumcision site. However, blood loss can be significant.

The mortality rate of classical VKDB is very low in developed countries. However, in Ethiopia, researchers reported a mortality rate of 25%. Mortality rates from classical VKDB are probably higher in developing countries such as Ethiopia because of scarce medical resources and a delay between when babies start to bleed and when they receive treatment (Lippi & Franchini, 2011).

What is the treatment for Vitamin K deficiency bleeding?

The main treatment for VKDB is to give the infant Vitamin K. When an infant with VKDB receives a shot of Vitamin K1, this will usually slow or stop the bleeding within 20-30 minutes (Shearer 2009). However, if bleeding happens in the brain, the infant may already have brain damage by the time the shot is given.

Other treatments that have been used in infants with late VKDB include blood and plasma transfusions, brain surgery to remove the accumulated blood, and administration of anti-seizure medications (Personal communication, Dr. Robert Sidonio, 2014).

Delayed cord clamping raises iron levels, so wouldn’t it raise Vitamin K levels as well?

Delayed cord clamping raises iron levels because cord blood is rich in iron. In contrast, cord blood has extremely low levels of Vitamin K1 (<.05 micrograms per Liter). Vitamin K1 is poorly retained in the body, and the Vitamin K1 that is stored is primarily in the liver, not in the bloodstream. So although delayed cord clamping increases iron levels, it is highly unlikely that this would help raise Vitamin K levels enough to prevent VKDB (Shearer 2009; Olson 2000).

In one small study, researchers measured Vitamin K levels in nine expectant mothers and then measured the levels in the cord blood after birth.  These mothers were healthy and had normal Vitamin K levels, but there was no Vitamin K detected in the cord blood. Six other mothers received 1 mg of intravenous Vitamin K1 right before delivery. After these mothers were given Vitamin K, the researchers were able to detect Vitamin K1 in the cord blood in 4 out of 6 infants, but the levels were still very low. It is thought that Vitamin K1 either does not cross the placenta easily, or that the baby’s blood lacks enough fatty lipids that are needed to pick up the Vitamin K1 (Shearer, Rahim et al. 1982).

Why do breastfed babies not have enough Vitamin K?

There are two main reasons why babies do not have enough Vitamin K in their system.

First, babies are born with very limited amounts of Vitamin K. Their levels are lowest at days 2-3 and do not reach adult levels until about 6 months of age. The levels are low because:

  • Very little Vitamin K1 transfers from the mother to the baby through the placenta
  • Babies do not have enough bacteria in their intestines to make Vitamin K2.

Second, breast milk has very tiny amounts of Vitamin K. Colostrum has about 2 micrograms of Vitamin K per Liter, while mature milk has 1 microgram per Liter (von Kries et al, 1987).

Virtually all babies with late VKDB are exclusively breastfed. When studies looked closely at infants who develop late VKDB, they found that mothers of these babies had normal levels of Vitamin K in their milk supply. It is thought that maybe some of these babies had a problem with absorbing the Vitamin K from their mother’s milk (Shearer 2009).

Why are formula-fed babies protected from Vitamin K deficiency bleeding?

There are virtually no reports of VKDB occurring in infants who are formula fed. This is because in contrast to breast milk, formula has relatively high levels of Vitamin K1—55 micrograms per liter (Shearer 2009).

On average, babies who are fed formula receive nearly 100 times more Vitamin K1 than babies who are breastfed (45.4 micrograms per day compared to 0.55 micrograms per day). Blood levels of Vitamin K1 in 6-week old breastfed babies are about 0.13 micrograms per liter, compared to 6.0 micrograms per liter in formula-fed babies (von Kries, Shearer et al. 1987Greer, Marshall et al. 1991Shearer 2009).

How much Vitamin K is in the shot and how much might actually be needed to produce the desired result?

Vitamin K1 injections are made under several brand names:

  • Phylloquinone ®
  • Phytonadione ®
  • AquaMEPHYTON ®
  • Mephyton ®
  • Konakion ®

(Lippi and Franchini, 2011).

Depending on the country in which the Vitamin K is administered, there are 1 to 2 mg of Vitamin K1 in the injection. This dose is what was tested in the original studies on Vitamin K, starting in the 1960s and continuing into the 1990s. Although this amount may seem high to some, it is thought that the Vitamin K1 injection is temporarily stored in the leg muscle and gradually released into the baby’s system over the next several months. This kind of delayed-release explains why the shot protects babies from both classic AND late Vitamin K deficiency bleeding (Loughnan and McDougall 1996) .

What are the ingredients in the shot?


A Vitamin K injection and packaging


It has been reported that some parents refuse the injection because they are concerned about ingredients in the shot, which the parents call “toxins.” One way to alleviate this concern is to ask your hospital if they have the preservative-free version of Vitamin K.

The ingredients in a shot with NO preservatives include:

  • 1 mg of Vitamin K1, a fat-soluble vitamin derived from plants
  • 10 mg of Polysorbate 80, which helps Vitamin K1 (a fat-soluble Vitamin) dissolve in liquid for the injection. Polysorbate 80 is made from natural sorbitol and plant-based oleic acid, is used in a wide variety of foods, medicines, and vitamin supplements, and is included in the Handbook of Green Chemicals.
  • 10.4 mg of Propylene glycol, which helps absorb extra water and maintain moisture in certain medicines. Propylene glycol has been recognized as safe by the FDA for use in food products.
  • 0.17 mg of Sodium acetate anhydrous, a mixture of salt and bicarbonate, that is used to adjust the pH of the injection
  • 0.00002 mL of Glacial acetic acid, also known as vinegar, that is used to adjust the pH of the injection

Does circumcision increase the need for Vitamin K injection?

Circumcision sites are frequently listed as a site of bleeding when infants have classical (first-week) VKDB. Unfortunately, circumcision often takes place when Vitamin K levels in the infant are lowest—during days 2 and 3 of life (Shearer 2009).

Infants who are circumcised and whose parents decline Vitamin K may be more likely to experience bleeding at the circumcision site, especially if the baby is breastfed. In a large clinical trial in the 1960s, researchers found that administering Vitamin K at birth can decrease the risk of bleeding during a circumcision. In this study, infants who were born on odd-numbered days received a Vitamin K shot at 24 hours of age, while infants who were born on even-numbered days did not. Bleeding occurred after circumcision in 6 out of 240 infants (2.5%) who received Vitamin K, and 32 out of 230 infants (13.9%) who did not have the Vitamin K shot (Vietti, Stephens et al. 1961).

A case report was recently published where an infant in a circumcision trial in Africa developed bleeding two hours after his circumcision. After applying pressure for 90 minutes, with no decrease or cessation in the bleeding, they discovered that the baby had not received a Vitamin K injection. He received a 2-mg injection and the bleeding stopped within 30 minutes (Plank, Steinmetz et al. 2013)

Does eating a maternal Vitamin K rich diet during pregnancy and nursing help Vitamin K levels in newborns?


On the fourth day after surgery, Olive's breathing tube came out. Here is Olive and her mother, Leona. Leona says "I have never been happier to hear a baby cry."


There is no good evidence that giving the mother extra Vitamin K during pregnancy can prevent VKDB in infants. In the largest known study looking at diets and Vitamin K deficiency, researchers followed 683 mothers before pregnancy and after giving birth. Blood was drawn from mothers during labor and from the umbilical cord after birth.  Mothers were asked about food intake during pregnancy and also interviewed by a dietitian during the postpartum period.  Researchers found no relationship between the Vitamin K status of mothers and that of their infants (Chuansumrit, Plueksacheeva et al. 2010).

Some people have suggested that an alternative strategy for boosting the Vitamin K intake of breast fed babies is for the mother to take a daily supplement herself after birth. However, there is very little evidence supporting this strategy. In one small study with only 6 mothers, a 2.5 mg oral dose twice a day (one hundred times the amount that would otherwise need to be given to the baby each day) was enough to raise the vitamin content of the milk to acceptable levels (Bolisetty, Gupta et al. 1998). In a large Japanese study with more than 3,000 mother-infant pairs, researchers tested a maternal dose of 15 mg of Vitamin K2 by mouth once a day. They found that this dose resulted in low infant Vitamin K levels in only 0.11% of the treatment group.  (Nishiguchi, Saga et al. 1996). But so far, researchers have not tested the effects of maternal Vitamin intake on actual Vitamin K deficiency bleeding in infants.

Why is the Vitamin K1 injection the preferred method in the U.S.?

The Vitamin K1 injection, given as a shot in the muscle (IM = intramuscular) is the preferred method for several reasons (Puckett and Offringa 2000Shearer 2009):

  • The shot is absorbed more easily than the oral version.
  • The shot has a delayed release effect that protects against both classical and late bleeding.
  • When the shot is used, the chance of late VKDB is near zero (does not completely eliminate the risk in cases of underlying liver or gallbladder disorders)
  • In contrast, oral Vitamin K1 lowers the chance of VKDB but does not eliminate it entirely. Also, infants with underlying (and sometimes undetected) gallbladder or liver disorders may not be able to absorb the oral Vitamin K

One reason that other countries may use the oral version of UK is that mothers and infants either have prolonged hospital stays after birth, or they have nurses come to the home. This does not occur in the U.S., and if the oral version is used, the parents need a reminder to administer the follow-up doses, and someone needs to monitor that the infant does not spit it up. When oral Vitamin K is used it usually requires 3 doses (birth, 1 week, and 6 weeks), and the breakthrough cases of Vitamin K deficiency bleeding are often related to missing the final dose (Busfield, Samuel et al. 2013).

If all infants are born with low Vitamin K levels, is it really a deficiency or is this the natural design of human beings?

Why are babies born with insufficient Vitamin K?  Obviously, it is impossible for us to knowwhy this happens. There are a couple of possibilities, and there isn’t really much research to inform this…but here are a few theories:

  1. VKDB, although catastrophic when it happens, is rare. So if you are looking at this as a case of “survival of the fittest,” perhaps there is not a pressing need for newborns to be born with higher levels of Vitamin K.
  2. When infants are born, many of their systems are not fully developed yet. For example, their nervous system and immune system are immature. It is possible that maybe an infant’s clotting system also needs time to mature and come into its full strength.
  3. Perhaps there is a reason we don’t know of that leads to low transmission of Vitamin K from mom to baby before and after birth. Maybe there is an unknown beneficial mechanism that is preventing some kind of environmental toxin from reaching the baby, and this mechanism also has the side effect of keeping Vitamin K from reaching baby in sufficient quantities through the placenta and breastmilk.

You could also make the argument that it doesn’t really matter why babies are born with low levels. The point is that they are born with low levels of Vitamin K, and that some babies will die from Vitamin K deficiency bleeding if they do not receive supplemental Vitamin K at the beginning of life. Most will not bleed. But some will, and some will experience brain injury or death. And these injuries and deaths are 100% preventable.

Are there any other risk factors for late VKDB, aside from breastfeeding?

The two main risk factors for VKDB are exclusive breastfeeding and not receiving the Vitamin K shot.  Virtually all cases of VKDB happen in infants who are exclusively breastfed and who have not received the shot.

It is important to note that Vitamin K deficiency bleeding can happen to any infant, whether they are pre-term and full-term, male or female, trauma or no trauma. Researchers have not been able to identify exactly which infants are at highest risk. Because of this, doses of Vitamin K are typically given to all newborns or to those whose mothers intend to exclusively breastfeed (Shearer, 2009).

Do you need Vitamin K if you have a trauma-free birth? Do we perceive a need for Vitamin K simply because it was studied during years of operative vaginal deliveries (forceps, vacuum) when babies were cord clamped immediately and taken away from moms with no breastfeeding all?

There is simply no evidence to support this theory.

In 1944, researchers who introduced the Vitamin K shot found that babies may be more likely to have brain bleeds or intestinal bleeds shortly after birth if they had a traumatic birth (Lehmann, 1944). However, this 60-year old finding has evolved into a myth in which people believe that the only babies who are at risk are those who had traumatic births (Cesarean section, forceps, or vacuum delivery). I have seen this myth perpetuated in many blog articles and on social media, without any supporting evidence or references to back up these claims.

In fact, I could find no recent evidence supporting the theory that infants born with instrumental help, or by Cesarean, are at higher risk for Vitamin K deficiency bleeding.

In one of the largest studies on this topic, Thai researchers followed women during pregnancy and after birth and examined risk factors for Vitamin K deficiency. The researchers labeled infants as “high risk” for Vitamin K deficiency if they were small for gestational age, born pre-term, or were born by Cesarean or forceps/vacuum delivery. The researchers found no difference in the percentage of infants with Vitamin K deficiency between the low-risk and high-risk infants (Chuansumrit et al, 2013).

As far as insufficient breastfeeding in the first hours of life goes, there is a link between insufficient amounts of breast milk in the first few days of life and classical VKDB. And it is theoretically possible that traumatic experiences at birth would make a baby more likely to bleed during the first week of life if they have low Vitamin K levels. But research (see above) has not supported the theory that traumatic deliveries are directly related to Vitamin K deficiencies.

Also, this theory does not explain late VKDB, which happens a week or more after birth, and is the more dangerous kind of bleeding. Late VKDB occurs too late to be impacted by the type of delivery.

Finally, as mentioned earlier, the timing of cord clamping probably does not have any effect on VKDB, since Vitamin K is usually undetectable in cord blood.

In summary, the most important risk factors for VKDB are exclusive breastfeeding and not getting the Vitamin K shot. Evidence does not support the claim that trauma at birth has anything to do with VKDB.

What is the exact number of lives saved with the Vitamin K shot?

The statistics vary from country to country. In Asian countries, where rates of VKDB are high, if you administer the Vitamin K1 shot to 100,000 infants, researchers estimate this would prevent 11 deaths, 340 years of lost life from the children who would have died, and 53 cases of life-long disability (Danielsson, Hoa et al. 2004).

In European countries, when they went from no Vitamin K1 to giving Vitamin K1 shots, researchers estimate that this probably prevented anywhere from 4 to 7 cases of late VKDB per 100,000 infants (Shearer, 2009).

Is there a risk of leukemia associated with injection?

The short answer is no.

The long answer is that this is a really interesting story:

In 1990, a British newspaper reported that researchers had found a link between Vitamin K injections and childhood leukemia (Golding, Greenwood et al. 1992). When the study was published, there was a massive switch in Great Britain from the Vitamin K shot to oral Vitamin K. There was also a huge rush to study Vitamin K1 injections to find out whether there really was a link between the injection and cancer (Shearer 2009).

Over the next two decades, there were a total of 12 studies examining the link between injectable Vitamin K and leukemia. Out of these 12 studies, two small studies (one being the original study) found a link. The other 10 studies found no relationship between Vitamin K and childhood cancer.

In 1999, the World Health Organization convened a working group to look at the issue. After carefully reviewing the evidence, they issued a statement saying that there was not enough evidence to support a link between Vitamin K and childhood cancer.

The two highest- quality studies on this issue were published in 2002 and 2003:

  • In 2002, researchers combined data from six major studies that looked at the potential relationship between Vitamin K and childhood cancer. There were 2,431 children with cancer and 6,338 children without cancer in these studies. The researchers found no association between injectable Vitamin K and any type of childhood cancer (Roman, Fear et al. 2002).
  • In 2003, researchers in Great Britain conducted the highest-quality study to date to determine whether there was a relationship between Vitamin K and childhood cancer. In this study, there were 2,530 children with cancer (half of whom had leukemia) and 4,487 children without cancer. The researchers found that 39% of children with cancer had received the injectable Vitamin K, while 42% of children without cancer had received the Vitamin K. The researchers stated that “there is no convincing evidence that neonatal Vitamin K administration influences the risk of children developing leukemia or any other cancer” (Fear, Roman et al. 2003)

After twenty-four years of studying the possibility of a link between Vitamin K and childhood cancer, researchers have now come to the conclusion that there is no evidence supporting a relationship between Vitamin K and leukemia or other childhood cancers (Shearer, 2009).

However, these fears still persist in the general public. In fact, some of the children in Tennessee who developed life-threatening VKDB did not receive Vitamin K because their parents thought that the shot could cause leukemia. This is not surprising, given that rumors, myths, and non-evidence based information about Vitamin K run rampant on the internet.

For example, I found a 2013 blog article on a popular natural health website, claiming that your child’s chance of getting leukemia from the Vitamin K shot is 1 in 500. The same article, as well as others, lists the side effects of intravenous Vitamin K and state that your child is at risk for these side effects if they receive the Vitamin K shot. These authors do not even understand the difference between intravenous and intramuscular injections. And yet parents all over the world are reading information from these websites and making important health decisions based on misleading and inaccurate claims.

Please do not make any health decisions for yourself or your family based on information you read on the internet (including this article) without talking with your healthcare provider first.

Is anyone keeping nationwide stats on how many infants get Vitamin K, and how many infants develop bleeds?

The U.S. is not tracking this data.

Some countries such as the United Kingdom, Germany, and the Netherlands have surveillance programs to monitor the effectiveness. (See Table below)

What is the evidence for the oral and injectable versions of Vitamin K? Are they effective?


K-Quinone is sold in the U.S. as an oral Vitamin K supplement. It has not gone through the FDA approval process.


In 2009, researchers published a Cochrane review combining the results of 13 studies that randomly assigned infants—almost all of whom were born at term and without complications—to either oral or injectable Vitamin K. Because late VKDB is so rare, the researchers could only look at the effects of Vitamin K on classical VKDB and laboratory results (Puckett and Offringa 2000).

Most of the studies in the Cochrane review looked only at infants who were exclusively breastfed. Two of the studies were done in the 1960s, and the rest took place between 1985 and 1996.

What treatments did the researchers study?

  • Seven studies compared one dose of oral Vitamin K to one dose of injectable Vitamin K. Doses ranged from 1-6 mg and were given within 12 hours of birth.
  • One study compared three doses of oral Vitamin K to one dose of injectable Vitamin K.
  • Four studies compared one dose of oral Vitamin K to nothing or placebo.
  • Four studies compared one dose of injectable Vitamin K to nothing or placebo.

None of the studies specifically looked to see whether there were any side effects.

In two very important studies that took place in the 1960s, researchers compared injectable Vitamin K to no Vitamin K for the prevention of classic VKDB. The researchers found that Vitamin K led to a 27% decrease in the risk of bleeding between one and seven days, and an 82% decrease in the risk of bleeding after a circumcision.

In the other studies, most of the researchers found no cases of bleeding. This is because VKDB is such a rare outcome that it is hard to study in a clinical trial. Instead, researchers looked at changes in laboratory results that indicate Vitamin K deficiency.

Multiple researchers found that giving Vitamin K1—whether it was a shot or an oral dose—significantly improved the baby’s lab results in the first week of life, when compared to nothing or a placebo.

In the studies that compared the Vitamin K shot to a single dose of oral Vitamin K, some researchers found no difference in lab results. However, when researchers looked specifically at Vitamin K levels, they found that the Vitamin K shot resulted in significantly higher levels of Vitamin K at one week and one month when compared to the single oral dose.

Is there any evidence that Vitamin K can prevent late Vitamin K deficiency bleeding?

Because late VKDB is such a rare event, all of the clinical trials in the Cochrane review were too small to determine if Vitamin K can prevent late VKDB.  In fact, it would take 700,000 infants per treatment group—1.4 million infants in total—to determine a difference in late VKDB. This kind of trial is logistically impossible to carry out (Cornelissen, von Kries et al. 1997).

So instead of looking at clinical trials, we need to review studies that compare regimens in different time periods. For example, you could compare rates of VKDB during time periods when countries used nothing, when they used oral Vitamin K, and when the Vitamin K shot became standard care.

Table 2 below shows the rates of late VKDB in multiple countries over different time periods. As you can see, the Vitamin K shot (IM Vitamin K1) basically eliminated all cases of late VKDB. The use of three doses of oral Vitamin K1 (at birth, one week, and one month of age) lowered the risk of late VKDB but did not eliminate it entirely.

Table 2: Rates of late Vitamin K deficiency bleeding in infants with no Vitamin K, oral Vitamin K, and Vitamin K shots

Vitamin K table

IM = Intramuscular (Vitamin K shot). MK4 is a type of Vitamin K2 that is not available in most countries
Data from the UK, Sweden, Switzerland (1986-1988), Germany (1988-1989), Japan, and Thailand were compiled in one paper by Shearer et al., 2009; Data from Australia, the Netherlands, Germany (1993-1994), and Switzerland (1995) compiled in one paper byCornellisen et al. (1997).

Why does oral Vitamin K sometimes fail to prevent bleeds?

Let’s look at Germany as an example of how oral Vitamin K can sometimes fail to prevent bleeds (see Table 2). In 1993-1994, Germany was using a 3-dose oral regimen of Vitamin K. In the 3-dose regimen, infants received 1 mg of Vitamin K orally 3 times—at birth, 1 week, and 2-3 months. During this time, Germany had 32 cases of VKDB out of 1.2 million births (for an incidence of 2.7 per 100,000) (von Kries, Hachmeister et al. 1995).

Out of these 32 cases, 2 infants did not receive any Vitamin K at all, 6 infants did not complete the entire 3-dose regimen, and 22 received all 3 doses of oral Vitamin K. This means that the 3-dose oral regimen—even when given in its entirety—still failed some of the time.In other words, although giving oral Vitamin K1 is better than nothing, it does not work 100% of the time, and infants who receive the oral regimen are still at risk for late VKDB.

If parents choose the oral version of Vitamin K, it is very important that they give their infant all three doses. However, even if all three doses are completed, the infant will still be at risk for bleeding.

On the other hand, almost every research study has shown that giving the Vitamin K shot works nearly 100% of the time at protecting infants from late VKDB.

What about using daily oral Vitamin K instead of the 3-dose regimen?

There have been no clinical trials that have compared daily oral Vitamin K to the Vitamin K shot.Some countries are using a daily regimen of oral Vitamin K1, instead of the 3-dose regimen shown in the table above.

For example, in the Netherlands, infants receive 1 mg of oral Vitamin K1 at birth, and then 25 mcg of Vitamin K1 daily by mouth, starting at week 1 and going through week 13.

There was some initial evidence from observational studies that giving oral Vitamin K to breastfed infants either weekly (1 mg per week) or daily (25 mcg per day) may be as effective as the single shot. This amount of Vitamin K would be similar to what formula fed babies are receiving on a daily basis.

However, it is important to remember that when this fat-soluble vitamin is given on an empty stomach, it may not be absorbed as well as the Vitamin K1 that is mixed into formula (Cornelissen, Kollee et al. 1993Cornelissen, von Kries et al. 1997).

Another problem with the daily or weekly regimen is compliance—a significant number of parents are not going to remember to give a daily vitamin for 13 straight weeks, no matter how well-intentioned they are.

In 2005, the Dutch started seeing some cases of late VKDB, so they did some research to evaluate the daily oral regimen. The results showed that the oral regimen did not protect infants with gallbladder disease, which is why they were seeing infants with late VKDB (Ijland, Pereira et al. 2008; van Hasselt, de Koning et al. 2008)

Is it possible to buy oral Vitamin K for infants?

Many research articles say that oral Vitamin K1 is not available in the U.S. That is because there is not an FDA-approved oral version for sale.However, there is an oral Vitamin K1 supplement, called K-Quinone (Scientific Botanicals, Inc) that you can buy online in the U.S.

K-Quinone is made up of Vitamin K1 from alfalfa, nettles, and green tea. This supplement is diluted in an olive and soy oil base. Because it is sold as a supplement without FDA approval, this medication is not required to have the stated amount of vitamin K. The amount could vary widely from vial to vial, and K-Quinone is not regulated or certified by a third party.

What are the pros of the Vitamin K shot?

  • Eliminates the possibility of classic and late VKDB.
  • Vitamin K is slowly released over time from the injection site, thus providing sufficient Vitamin K1 until the baby’s Vitamin K levels reach adult levels naturally.

What are the cons of the Vitamin K shot?

  • Causes pain, which can be minimized by having the infant breastfeed while the shot is given.
  • Can cause bleeding or bruising at the injection site.

What are the pros of oral Vitamin K?

  • Easy to give and not invasive.
  • Lowers the risk of classic and late VKDB… but does not eliminate the risk entirely.

What are the cons of oral Vitamin K?

  • Some babies may not be able to absorb it, or they may spit it up.
  • If the baby has undetected gallbladder or liver disease, the oral Vitamin K will not protect them from VKDB.
  • Not 100% effective at preventing VKDB (although it is better than nothing).
  • Requires at least three doses and parents may forget to complete the regimen.

Pros and Cons taken from the Cochrane review (Puckett and Offringa, 2000)

What are the current recommendations for giving Vitamin K?

The American Academy of Pediatrics states: “Vitamin K1 should be given to all newborns as a single, intramuscular dose of 0.5 to 1 mg.”  They say that it is okay to delay administration of intramuscular Vitamin K until after the first feeding is completed, but that it should be given within six hours of birth. The AAP does not recommend oral Vitamin K, because some infants may have trouble absorbing it and there is no FDA-approved version in the U.S.

In the Netherlands, the current practice recommendation consists of giving 1 mg Vitamin K orally directly after birth and a daily dose of 25 mcg from day 8 onwards.

In New Zealand, the guidelines state that infants should receive 1 mg of Vitamin K as a single intramuscular shot at birth. If parents do not consent to the shot but do consent to oral Vitamin K, then 2 mg of Vitamin K should be given by mouth soon after birth, once at 3-7 days, and again at 6 weeks. Parents should be informed that bleeding can still occur with the oral Vitamin, even if the parents comply with the oral regimen.

In the United Kingdom, guidelines state that all parents should be offered Vitamin K for their infants. The preferred way of giving Vitamin K is through a one-time shot shortly after birth. If the parents do not consent to the Vitamin K shot, they can be offered the oral Vitamin K, but they should be informed that this method requires multiple doses.

What’s the bottom line?

Olive surprised her physicians and made a miraculous recovery. Apart from the scar on her head, you would never even know she experienced a brain bleed. Olive's mother realizes that not everyone will accept the Vitamin K shot. But her hope is that when someone wants to say “No,” they will have seen what could happen to their child and maybe think twice.
Olive surprised her physicians and made a complete recovery. Here she is pictured with her older sister.

Giving a breastfed infant aVitamin K1 shot virtually eliminates the chance of life-threatening Vitamin K deficiency bleeding. The only known adverse effects of the shot are pain, bleeding, and bruising at the site of the injection.

A regimen of three doses of oral Vitamin K1 at birth, 1 week, and 1 month reduces the risk of bleeding. Although oral Vitamin K1 is better than nothing, it is not 100% effective. It is important for parents to administer all 3 doses in order for this regimen to help lower the risk of late Vitamin K deficiency bleeding.

Recently, there have been many myths, misconceptions, and misinformation floating around the internet and social media about Vitamin K. It is important that parents look at the facts so that their consent or refusal is informed. Right now, parents who have been declining Vitamin K may not have all the information, or they may have been given inaccurate information.


I would like to acknowledge Mandi Hardy Hillman, PhD, LPC, CD(DONA), ICCE, for her invaluable assistance with the literature search.

I would like to acknowledge my expert reviewers: Mark Sloan, MD, Pediatrician and Assistant Clinical Professor at the University of California, San Francisco, and Robert F. Sidonio, Jr., MD, Pediatric Hematologist-Oncologist and Assistant Professor at Vanderbilt University.

I would also like to thank Cristen Pascucci and Sharon Muza CD(DONA), BDT(DONA), LCCE, FACCE for their medical editing assistance.

Thank you to Stefani for sharing Olive’s story and photos.

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Read an interview with Rebecca about what it was like to write this article!

I shared my personal thoughts on the evidence for the Vitamin K shot in this exclusive interview with Lamaze’s research blog, Science and Sensibility. To read the interview, clickhere.

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American Academy of Pediatrics. (1961). “Report on Committee on Nutrition: Vitamin K Compounds and the Water-Soluble Analogues.” Pediatrics28: 501-507. Free full text, clickhere.

American Academy of Pediatrics (2003). “Controversies concerning vitamin K and the newborn. American Academy of Pediatrics Committee on Fetus and Newborn.”Pediatrics112(1 Pt 1): 191-192. Click here.

Bolisetty, S., J. M. Gupta, et al. (1998). “Vitamin K in preterm breastmilk with maternal supplementation.” Acta Paediatr87(9): 960-962. Click here.

Busfield, A., R. Samuel, et al. (2013). “Vitamin K deficiency bleeding after NICE guidance and withdrawal of Konakion Neonatal: British Paediatric Surveillance Unit study, 2006-2008.” Arch Dis Child98(1): 41-47. Click here.

Chuansumrit, A., T. Plueksacheeva, et al. (2010). “Prevalence of subclinical vitamin K deficiency in Thai newborns: relationship to maternal phylloquinone intakes and delivery risk.” Arch Dis Child Fetal Neonatal Ed95(2): F104-108. Click here.

Cornelissen, E. A., L. A. Kollee, et al. (1993). “Prevention of vitamin K deficiency in infancy by weekly administration of vitamin K.” Acta Paediatr82(8): 656-659. Click here.

Cornelissen, M., R. von Kries, et al. (1997). “Prevention of vitamin K deficiency bleeding: efficacy of different multiple oral dose schedules of vitamin K.” Eur J Pediatr156(2): 126-130. Click here.

Danielsson, N., D. P. Hoa, et al. (2004). “Intracranial haemorrhage due to late onset vitamin K deficiency bleeding in Hanoi province, Vietnam.” Arch Dis Child Fetal Neonatal Ed89(6): F546-550. Free full text, click here.

Fear, N. T., E. Roman, et al. (2003). “Vitamin K and childhood cancer: a report from the United Kingdom Childhood Cancer Study.” Br J Cancer89(7): 1228-1231. Free full text, click here.

Golding, J., R. Greenwood, et al. (1992). “Childhood cancer, intramuscular vitamin K, and pethidine given during labour.” Bmj305(6849): 341-346.

Greer, F. R., S. Marshall, et al. (1991). “Vitamin K status of lactating mothers, human milk, and breast-feeding infants.” Pediatrics88(4): 751-756. Click here.

Hanawa, Y., M. Maki, et al. (1988). “The second nation-wide survey in Japan of vitamin K deficiency in infancy.” Eur J Pediatr147(5): 472-477. Click here.

Ijland, M. M., R. R. Pereira, et al. (2008). “Incidence of late vitamin K deficiency bleeding in newborns in the Netherlands in 2005: evaluation of the current guideline.” Eur J Pediatr167(2): 165-169. Free full text, click here.

Lehmann, J. (1944). “Vitamin K as a prophylactic in 13,000 infants.” Lancet243(6294): 493-494. Click here.Lippi, G. and M. Franchini (2011). “Vitamin K in neonates: facts and myths.”Blood Transfus9(1): 4-9. Free full text, Click here.

Loughnan, P. M. and P. N. McDougall (1996). “Does intramuscular vitamin K1 act as an unintended depot preparation?” J Paediatr Child Health32(3): 251-254. Click here.

Nishiguchi, T., K. Saga, et al. (1996). “Vitamin K prophylaxis to prevent neonatal vitamin K deficient intracranial haemorrhage in Shizuoka prefecture.” Br J Obstet Gynaecol103(11): 1078-1084.

Olson, R. (2000). Vitamin K. Modern nutrition in health and disease. M. Shils, J. Olson and M. Shike. Philadelphia, Lippincott363.

Plank, R. M., T. Steinmetz, et al. (2013). “Vitamin K deficiency bleeding and early infant male circumcision in Africa.” Obstet Gynecol122(2 Pt 2): 503-505. Click here.

Polin, R. A., W. W. Fox, et al. (2011). Fetal and neonatal physiology, Saunders. 40. Click here.Puckett, R. M. and M. Offringa (2000). “Prophylactic vitamin K for vitamin K deficiency bleeding in neonates.” Cochrane Database Syst Rev(4): CD002776. Click here.

Roman, E., N. T. Fear, et al. (2002). “Vitamin K and childhood cancer: analysis of individual patient data from six case-control studies.” Br J Cancer86(1): 63-69. Free full text, clickhere.

Shearer, M. J. (2009). “Vitamin K deficiency bleeding (VKDB) in early infancy.” Blood Rev23(2): 49-59. Click here.Shearer, M. J., S. Rahim, et al. (1982). “Plasma vitamin K1 in mothers and their newborn babies.” Lancet2(8296): 460-463. Click here.

van Hasselt, P. M., T. J. de Koning, et al. (2008). “Prevention of vitamin K deficiency bleeding in breastfed infants: lessons from the Dutch and Danish biliary atresia registries.”Pediatrics121(4): e857-863. Click here.Vietti, T. J., J. C. Stephens, et al. (1961). “Vitamin K-1 prophylaxis in the newborn.” JAMA176: 791-793. Click here.

von Kries, R., A. Hachmeister, et al. (1995). “Repeated oral vitamin K prophylaxis in West Germany: acceptance and efficacy.” BMJ310(6987): 1097-1098. Click here.

von Kries, R., M. Shearer, et al. (1987). “Vitamin K1 content of maternal milk: influence of the stage of lactation, lipid composition, and vitamin K1 supplements given to the mother.”Pediatr Res22(5): 513-517. Click here.

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Grieving for birth is selfless not selfish


Interesting reading …

From the webpage … click link to read full article.

The term stolen birth refers to any birth that hasn’t gone as the mother planned, some birth is stolen by iatrogenic hospital interventions, and some when nature goes awry and unplanned interventions become necessary. The bottom line is that a woman feels like she lost the birth she dreamed of, her birth has been stolen. 

Women grieve stolen birth experiences very deeply, but their grief often remains private because modern birth culture maintains that a healthy baby is the one and only goal. The roots of “the healthy baby lie” are found in the reality of birth, that the outcome is unknown and one potential outcome is, quite undeniably, death. But to women, birth means a great deal more than being alive afterwards. Birth is the introduction to their baby, it matters a great deal.


What is physiological birth?


Interesting read … here’s the start click the link for the rest …

Natural physiological birth is an involuntary process orchestrated by neurochemicals and hormones that unfurl a woman’s body, enabling her to give birth to a baby and make the transition to motherhood. Physiological birth is directed from within the limbic system of the brain and body in response to and

The First Cry
The First Cry

in collaboration with the endocrine system of the baby and placenta. This allows a woman to respond and surrender to ancient primal patterns and physical instincts. It can occur best when a mother is not disturbed during the process of labour and birth. Giving birth physiologically is not easy in our highly medicalized and fear driven cultural climate.

Why your baby needs 40 weeks …


From the webpage:  

In recent years, some women have been electing to have their babies born early for non-medical reasons, such as convenience or fear of a big baby. This worrying trend leads to a plethora of increased risks and health problems for both baby and mother – something you don’t want to be worrying about during the birth, or recovering from after the birth. The babymoon should be a special, bonding time with lots of snuggles and cuddles, without worrying about pain or illness. If you think logically, in a week or two, your baby isn’t going to sprout an extra 5cms circumference on his head, nor add an extra kilo of weight. A week will make little difference in terms of size, and no matter if you have a big baby or a small baby, women can have difficult and easy births. So many factors come into play, but if you’ve done some good research, you would know a thing or two about how to labour in a way to make it easiest for your body and baby – no matter what baby’s size might be.


When love feels heavy


From the webpage …. makes an interesting read … enjoy 

Before I was a parent, I was the perfect one. People told me my life would change. People told me I would be tired. That parenthood would be the greatest and hardest thing I would ever do.

Yeah yeah yeah.
I know. I know.
I knew everything.

My family would just smile and nod at my ignorance, and I wonder now if they were scared for me.

I recently sat in a friend’s baby shower. I was surrounded by women making light hearted jokes about new parenthood, about sleep depravation, and pregnancy cravings. They exchanged recommendations for swaddle blankets and butt creams. Underneath the small talk and “oohing” and “ahhing” over tiny gifted baby clothes, sat the realness, the hardness of motherhood. I could feel that every mom in the room, behind their sleepless sunken eyes, knew what that meant; they had felt that weight, but they only had the heart to give gifts and hugs and congratulations. I sat there in silence, when all I wanted to do was talk and talk and talk about how new motherhood really can be. To let her in on all the real secrets of being a mother.